Difference between revisions of "Team:UCL/Lungs"

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<p> References </p>
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[1] Bargagli, E., Olivieri, C., Bennett, D., Prasse, A., Muller-Quernheim, J. and Rottoli, P. (2009). Oxidative stress in the pathogenesis of diffuse lung diseases: A review. Respiratory Medicine, 103(9), pp.1245-1256. </li>
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<li> [2] FATTMAN, C., SCHAEFER, L. and OURY, T. (2003). Extracellular superoxide dismutase in biology and medicine. Free Radical Biology and Medicine, 35(3), pp.236-256.</li>
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<li> [3] Medina, J. (1996). The clock of ages. Cambridge: Cambridge University Press (Page 258)</li>
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<li> [4] Ahmed MN, Suliman HB, Folz RJ, Nozik-Grayck E, Golson ML, Mason SN, Auten RL. Extracellular superoxide dismutase protects lung development in hyperoxia-exposed newborn mice. Am J Respir Crit Care Med 167: 400–405, 2003</li>
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<li> [5] Auten RL, O'Reilly MA, Oury TD, Nozik-Grayck E, Whorton MH. Transgenic extracellular superoxide dismutase protects postnatal alveolar epithelial proliferation and development during hyperoxia. Am J Physiol Lung Cell Mol Physiol 290: L32–L40, 2006.</li>
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<li> [6]  Ghio AJ, Suliman HB, Carter JD, Abushamaa AM, Folz RJ. Overexpression of extracellular superoxide dismutase decreases lung injury after exposure to oil fly ash. Am J Physiol Lung Cell Mol Physiol 283: L211–L218, 2002.</li>
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<li> [7]  Dahl M, Bowler RP, Juul K, Crapo JD, Levy S, Nordestgaard BG. Superoxide dismutase 3 polymorphism associated with reduced lung function in two large populations. Am J Respir Crit Care Med 178: 906–912, 2008.</li>
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<li> [8] Juul K, Tybjaerg-Hansen A, Marklund S, Lange P, Nordestgaard BG. Genetically increased antioxidative protection and decreased chronic obstructive pulmonary disease. Am J Respir Crit Care Med 173: 858–864, 2006.</li>
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<li> [9] Wilk JB, Walter RE, Laramie JM, Gottlieb DJ, O'Connor GT. Framingham Heart Study genome-wide association: results for pulmonary function measures. BMC Med Genet 8, Suppl 1: S8, 2007.</li>
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<li> [10]  Daniel Jane-Wit, H. (2012). Special Issue on The Aging Lung: Mechanisms of Dysfunction in Senescent Pulmonary Endothelium. The Journals of Gerontology Series A: Biological Sciences and Medical Sciences, [online] 67A(3), p.236. Available at: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3297765/ [Accessed 18 Aug. 2016].</li>
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<li> [11] Rebecca E. Oberley-Deegan, J. (2009). Extracellular Superoxide Dismutase and Risk of COPD. COPD, [online] 6(4), p.307. Available at: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4075061/ [Accessed 18 Aug. 2016]. </li>
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Revision as of 18:28, 29 September 2016

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UCL iGEM 2016 | BioSynthAge

References

  • [1] Bargagli, E., Olivieri, C., Bennett, D., Prasse, A., Muller-Quernheim, J. and Rottoli, P. (2009). Oxidative stress in the pathogenesis of diffuse lung diseases: A review. Respiratory Medicine, 103(9), pp.1245-1256.
  • [2] FATTMAN, C., SCHAEFER, L. and OURY, T. (2003). Extracellular superoxide dismutase in biology and medicine. Free Radical Biology and Medicine, 35(3), pp.236-256.
  • [3] Medina, J. (1996). The clock of ages. Cambridge: Cambridge University Press (Page 258)
  • [4] Ahmed MN, Suliman HB, Folz RJ, Nozik-Grayck E, Golson ML, Mason SN, Auten RL. Extracellular superoxide dismutase protects lung development in hyperoxia-exposed newborn mice. Am J Respir Crit Care Med 167: 400–405, 2003
  • [5] Auten RL, O'Reilly MA, Oury TD, Nozik-Grayck E, Whorton MH. Transgenic extracellular superoxide dismutase protects postnatal alveolar epithelial proliferation and development during hyperoxia. Am J Physiol Lung Cell Mol Physiol 290: L32–L40, 2006.
  • [6] Ghio AJ, Suliman HB, Carter JD, Abushamaa AM, Folz RJ. Overexpression of extracellular superoxide dismutase decreases lung injury after exposure to oil fly ash. Am J Physiol Lung Cell Mol Physiol 283: L211–L218, 2002.
  • [7] Dahl M, Bowler RP, Juul K, Crapo JD, Levy S, Nordestgaard BG. Superoxide dismutase 3 polymorphism associated with reduced lung function in two large populations. Am J Respir Crit Care Med 178: 906–912, 2008.
  • [8] Juul K, Tybjaerg-Hansen A, Marklund S, Lange P, Nordestgaard BG. Genetically increased antioxidative protection and decreased chronic obstructive pulmonary disease. Am J Respir Crit Care Med 173: 858–864, 2006.
  • [9] Wilk JB, Walter RE, Laramie JM, Gottlieb DJ, O'Connor GT. Framingham Heart Study genome-wide association: results for pulmonary function measures. BMC Med Genet 8, Suppl 1: S8, 2007.
  • [10] Daniel Jane-Wit, H. (2012). Special Issue on The Aging Lung: Mechanisms of Dysfunction in Senescent Pulmonary Endothelium. The Journals of Gerontology Series A: Biological Sciences and Medical Sciences, [online] 67A(3), p.236. Available at: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3297765/ [Accessed 18 Aug. 2016].
  • [11] Rebecca E. Oberley-Deegan, J. (2009). Extracellular Superoxide Dismutase and Risk of COPD. COPD, [online] 6(4), p.307. Available at: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4075061/ [Accessed 18 Aug. 2016].